Dr. Anantha Harijith MD
Associate Professor of Pediatrics, University of Arizona College of Medicine, Tucson, USA Faculty, Division of Neonatology, University Hospitals Cleveland Medical Center, Cleveland, USA · United States
Editorial leadership for Journal of Current Viruses and Treatment Methodologies ISSN 2691-8862
Research interests
- Neonatal Lung Diseases Bronchopulmonary Dysplasia (Bpd) Neonatal Sepsis
- Inflammation Mitochondrial Dysfunction In Critical Illness Lysophosphatidic Acid Signaling Sphingolipid Biology Neonatal Airway Remodeling Translational Neonatology
Biography
Dr. Anantha K. Harijith is an Associate Professor of Pediatrics at the University of Arizona College of Medicine and a neonatology researcher at University Hospitals Cleveland. His research focuses on neonatal lung diseases, bronchopulmonary dysplasia, asthma, and sepsis-related inflammation. With extensive training in neonatology and clinical pediatrics, Dr. Harijith has contributed to translational studies investigating cellular mechanisms of neonatal respiratory disorders, sphingolipid signaling pathways, and mitochondrial dysfunction in critical illness. He collaborates on both clinical and pre-clinical studies aimed at improving outcomes in premature infants and children with chronic lung disease, publishing widely in respected peer-reviewed journals.
Education details:
- M.D., Medical Degree (Institution not specified in sources)
- MRCP (UK) – Royal College of Physicians, United Kingdom
Professional background:
- Associate Professor of Pediatrics, University of Arizona College of Medicine
- Faculty, Division of Neonatology, University Hospitals Cleveland Medical Center
- Research focus on neonatal lung injury, inflammation, and respiratory biology
Achievements:
- Authored 50+ peer-reviewed publications in neonatology and pediatric lung biology
- Recognized for translational research on bronchopulmonary dysplasia (BPD)
- Active member in pediatric and neonatal research consortia
Current research projects:
- Mechanisms of bronchopulmonary dysplasia in premature infants
- Sphingolipid and lysophosphatidic acid signaling in neonatal lung injury
- Mitochondrial dysfunction and immune dysregulation in neonatal sepsis
- Therapeutic targets in neonatal airway remodeling and inflammation
Selected publications
- The Role of Sphingolipid Signaling in Oxidative Lung Injury and Pathogenesis of Bronchopulmonary Dysplasia 2022 cited 26×
- Sphingosine kinase 1 regulates lysyl oxidase through STAT3 in hyperoxia-mediated neonatal lung injury 2021 cited 16×
- Genetic deletion of Sphk2 confers protection against Pseudomonas aeruginosa mediated differential expression of genes related to virulent infection and inflammation in mouse lung 2019 cited 14×
- Expression profiling of genes regulated by sphingosine kinase1 signaling in a murine model of hyperoxia induced neonatal bronchopulmonary dysplasia 2017 cited 14×
- Hyperoxia-induced S1P1 signaling reduced angiogenesis by suppression of TIE-2 leading to experimental bronchopulmonary dysplasia 2021 cited 11×
- Sphingosine kinase 1-specific inhibitor PF543 reduces goblet cell metaplasia of bronchial epithelium in an acute asthma model 2024 cited 7×
Ranked by citation impact (Crossref) where available, newest otherwise · verified via ORCID.
Considering JVAT for your work?
This journal is guided by Dr. Anantha Harijith (Associate Professor of Pediatrics, University of Arizona College of Medicine, Tucson, USA Faculty, Division of Neonatology, University Hospitals Cleveland Medical Center, Cleveland, USA ) and a peer-review board of practising researchers. Open access, author-retained copyright (CC BY), and a clear editorial process.